Complaints & connections · borters.ch

COPD

Bronchopneumopathia chronica obstructiva

DE FR EN

The limitation stays even on good days — and every flare-up costs lung function that does not come back.

Also known as: COPD · chronic obstructive pulmonary disease · smoker’s lung

Open in the tool — with search and all the links →

This compilation is for information and replaces neither medical diagnosis nor treatment. If you have symptoms, see a doctor.

A permanently narrowed and inflamed lung from which the air no longer comes out properly. That is exactly the difference from asthma, which comes and goes: here a limitation remains, even on good days. By far the commonest cause is years of smoking, then dust and fumes at work, smoke from open fires, and in younger people without that history an inherited lack of a protective protein. The onset is so unremarkable that the first years are nearly always missed: a morning cough put down to smoking, some sputum, and breathlessness that at first only shows uphill. Because one walks more slowly, it is noticed less — the illness is hidden by one’s own adaptation. The most important thing across the whole course are the flare-ups: each one costs lung function that does not come back, and many could be avoided. And the one lever larger than any medicine remains stopping smoking — the only thing shown to slow the decline, at every stage and at every age. One point is almost always understood the wrong way round: the feeling says no air is getting in — but the narrowing is on the way out. The lung normally recoils on its own like a rubber band; when that recoil is lost and the small airways collapse on breathing out, the old air stays in and the chest is already full. Whoever is full cannot breathe in. This is called hyperinflation, and it explains why slow, measured breathing out against almost closed lips does more in an attack than any effort to pull more air in. It is measured as the residual volume in lung function testing; how much oxygen actually crosses into the blood is told in addition by the diffusing capacity — low in emphysema, normal in asthma.

Levers & pathways

studiedStopping works at every stage

It is the only measure shown to slow the loss of lung function — even after forty years and even in advanced disease. Support and medication roughly double the chances.

studiedHave the inhaler technique checked

A large proportion of people use the device wrongly, and then even the best drug does nothing. Demonstrating it once a year is one of the most effective appointments there is.

studiedPulmonary rehabilitation is treatment

Supervised training improves capacity and quality of life more than most medicines. It works against the loop of breathlessness, taking it easy and muscle loss.

studiedHave a plan for the flare-up

In writing: how I recognise it, what I take then, when I call. That shortens flare-ups and prevents some hospital admissions.

studiedHave the oxygen measured

In advanced disease it is checked whether long-term oxygen is needed — used correctly it prolongs life, and that is a medical decision, not a matter of feeling. The threshold is a measured one: an oxygen pressure of 55 mmHg or below at rest, confirmed twice — or up to 60 mmHg if the blood is already thickened or the right heart is carrying the load. And the amount decides: the benefit is shown from at least fifteen hours a day, at night alone is not enough.

associatedKeep an eye on muscle and weight

Weight loss and muscle wasting measurably worsen the outlook. Protein and strength training therefore belong to the treatment and are not a side issue.

studiedRule out the threadworm before immune therapy

A threadworm can stay silent in the gut for decades and, under cortisone, biologicals or other immune suppression, flood lung and bloodstream. Anyone who ever went barefoot in the tropics or subtropics is looked for beforehand with a blood test — one effort, and it prevents the worst course this worm has.

studiedWhat the medicine does — and what it does not

The inhalers widen the airways and make breathing out easier. They hold the disease where it is; they do not rebuild the lung. What changes the course measurably is stopping smoking — and movement, because it trains the breathing muscles.

studiedNumbers and timeframes

About five to ten in a hundred people over forty are affected, and a large share do not know. The lung loses function with age anyway; with smoking it goes two to three times faster. After stopping, the loss returns to the normal age rate — function already lost does not come back, but the road ahead changes.

Possible complaints

associatedIt begins as a smoker’s cough

A morning cough with some sputum, dismissed as normal for years. Yet these are the years in which most could be saved.

associatedAt first only uphill

The breathlessness shows on slopes and stairs. Because one unconsciously slows down, the worsening is noticed late.

associatedBreathing out takes longer

That is the hallmark: air comes out with more difficulty than it goes in, often with a quiet wheeze and with pursed lips.

associatedThe flare-up changes everything

More sputum, a different colour, more breathlessness over days. Every flare-up costs substance, and counting them shows the course.

associatedWhat runs alongside

Muscle loss, weight loss, osteoporosis, heart failure and low mood often belong to it and are rarely treated alongside.

Blood values

associatedBlood count

A persistently low oxygen level raises the red cells. The eosinophils in turn help decide who benefits from certain inhalers.

studiedAlpha-1 antitrypsin

Measure once in a lifetime — especially with onset before fifty, with little or no smoking, and with cases in the family. It is the one cause with its own route.

infoCRP (inflammation)

Helps during a flare-up to separate a bacterial worsening from another cause — in everyday life it says little.

Nutrients

studiedVitamin D

Deficiency common; correcting may cut flares.

Supplements

studiedVitamin D

If deficient.

Hormones

associatedTestosterone

With advanced COPD the testosterone value in men is often low — through the illness itself and through repeated cortisone bursts. That contributes to the muscle wasting that shapes the course.

studiedParathyroid hormone (PTH)

Repeated cortisone bursts add up on bone. With COPD osteoporosis is therefore common and rarely looked for — bone density belongs in from several bursts a year onward.

associatedLeptin

With advanced disease weight falls although eating continues — the work of breathing itself uses energy, and inflammatory messengers dampen appetite. Weight loss here is a warning sign, not a success.

Food

studiedBalanced, preserve muscle

Underweight worsens outcomes.

Alternative approaches — what people use

studiedMovement & exercise

Pulmonary exercise improves capacity and quality of life more than any additional medicine.

studiedBreathing exercises

Pursed-lip breathing helps in the acute moment and is learned in minutes.

studiedDiet change

Being underweight worsens the course — enough protein is part of the treatment here.

TCM — how it is seen there

traditionalLung meridian

The Lung circle also covers the strength of the breath — in TCM the exhaustion of a long airway disease is placed there, not separately from it.

mixedQigong & Tai Chi

Of everything TCM offers, this has the best numbers: with COPD both measurably improve walking distance and breathlessness in studies. It is movement with breath guidance — and that is exactly how it works.

Frequencies — what is on file

traditionalCOPD (chronic obstructive pulmonary disease)

Listed values: 3 · 174 · 285 Hz